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19-Oct-2017 20:01

Significant changes were observed in the following areas: neurotransmitters, prostaglandins, nerve growth factor, Rho-kinase, interstitial cells of Cajal, and ion and transient receptor potential channels.Interestingly, alterations in these areas showed great variation in each of the conditions of the OAB, suggesting that the pathophysiology of the OAB might be different in each condition of the disease.When bladder volume reaches the micturition threshold, it activates the pontine micturition center to induce bladder contraction and relaxation of the urethra, leading to bladder emptying.

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According to the International Continence Society, OAB was defined as urgency, with or without urge incontinence, and usually with frequency and nocturia.

The neural circuitry that controls this process is complex; it involves pathways at many levels of the brain, spinal cord and the peripheral nervous system and is mediated by multiple neurotransmitters.